Ginkgo Biloba & Vitamin B-12
Neuro-Nutrient Formula with Ginkgo Biloba & Vitamin B-12
| $6.99 | 60 Count Bottle VP25A | Retails for $8.90 | |
| $69.99 | 12 Bottles 60 Count Each | Retail Value $106.80 | |
| $9.99 | 90 Count Bottle VP25P | Retails for $12.90 | |
| $18.79 | 180 Count Bottle VP25T | Retails for $23.90 | |
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Display An Attractive 12 Bottle Case On Your Counter for Patient/Client Convenience. Additional Wholesale Discounts Available |
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Nutritional research indicates that a significant relationship between intake of certain nutrients (including herbs, vitamins, minerals, amino acids) and proper functioning of the vascular system improves blood circulation to the brain.
Ginkgo Plus supplies a comprehensive range of these important nutrients in an easy-to-take, convenient formula for daily dietary supplementation.
This Exclusive Formula Has Been Developed Specifically With Key Neuro-Nutrients . . . In a natural base containing Ginkgo Biloba leaf powder, Alfalfa, Kelp, Lecithin, Parsley and Watercress.
No Sugar, Salt, Starch, Preservatives or Artificial Coloring Added.
Find Ginkgo Biloba Supplements at a Featured Merchant (below)
| Each 3 Tablet Contains | |
| Vitamin B-12 (Cobalamin Concentrate) | 1000 mcg |
| Choline (Choline Bitartrate) | 450 mg |
| Folic Acid | 400 mcg |
| L-Glutamine | 125 mg |
| L-Phenylalanine | 125 mg |
| Pitutary (freeze-dried glandular) | 15 mg |
| Potassium (Gluconate) | 99 mg |
| Ginkgo Biloba Leaf (with extract) | 40 mg |
| Soya Lecithin | 300 mg |
Ginkgo is one of the world's oldest tree species. Individual trees may live for as long as a thousand years. The last ice age almost eradicated Ginkgo but it survived in China and was brought to Europe in the 1700's. The tree is a widely used ornamental in parks and gardens throughout the world and is well-known for its golden leaves in the fall.
Pharmacologic ingredients in ginkgo biloba include flavonoid glycosides and terpenes. Although not fully understood, Ginkgo is used for its antioxidant effects and its ability to stimulate blood flow. Ginkgo has been utilized in Europe for many years for perceived circulatory system benefits, as well as mental function improvement.
New Study Conducted At Columbia University Medical Center Indicates Blood Sugar Linked To Normal Cognitive Aging...
Maintaining blood sugar levels, even in the absence of disease, may be an important strategy for preserving cognitive health, suggests a study published by researchers at Columbia University Medical Center (CUMC).
Those "senior moments" and common lapses in memory, could be blamed,at least in part, on rising blood glucose levels as people age. The findings suggest that exercising to improve blood sugar levels could be a way for some people to stave off the normal cognitive decline that comes with age.
This is news even for people without diabetes since blood glucose levels tend to rise as we grow older. Whether through physical exercise, diet or drugs, our research suggests that improving glucose metabolism could help some of us avert the cognitive slide that occurs in many of us as we age according to researchers from the department of neurology in the Sergievsky Center and in the Taub Institute for Research on Alzheimer's Disease and the Aging Brain at Columbia University Medical Center.
Although it is widely known that the early stages of Alzheimer's disease cause damage to the hippocampus, the area of the brain essential for memory and learning, studies have suggested that it is also vulnerable to normal aging. Until now, the underlying causes of age-related hippocampal dysfunction have remained largely unknown.
Previously, using high-resolution brain imaging, the team of researchers discovered that decreasing brain function in one area of the hippocampus, called the dentate gyrus, is a main contributor of normal decline in memory as people age. In this new study, funded by the National Institute on Aging (NIA), the American Diabetes Association and the McKnight Brain Research Foundation, the researchers mapped out the specific areas of the hippocampus impacted by late-life diseases like diabetes and stroke.
This research used imaging in both human volunteers and in animal models to help us better understand the basic mechanisms behind hippocampal dysfunction in the aged, While more research is needed into the complex interaction of late-life disease and how it may affect the hippocampus, this new study is part of an ongoing effort to identify specific areas whereinterventions might preserve cognitive health.
This new study looked at measures that typically change during aging, like rising blood sugar, body mass index, cholesterol and insulin levels. The research found that decreasing activity in the dentate gyrus only correlated with levels of blood glucose. Showing that blood glucose selectively targets the dentate gyrus is not only the most conclusive finding, but it is the most important for 'normal' aging- that is hippocampal dysfunction that occurs in the absence of any disease states.
Additional animal studies helped confirm the relationship between glucose and dentate gyrus activity. Interestingly, the researchers found the same association in aging rhesus monkeys and in mice. Preventing late-life disease would benefit the aging hippocampus and the findings suggest that maintaining blood sugar levels, even in the absence of diabetes, could help maintain aspects of cognitive health. More specifically, the new findings predict that any intervention that causes a decrease in blood glucose should increase dentate gyrus function and would therefore be cognitively beneficial.
The new findings also suggest that one way in which physical exercise could improve memory is via lowering glucose levels. Imaging studies in humans and in mice have documented that among all hippocampal subregions, physical exercise causes an improvement in dentate gyrus function.
By improving glucose metabolism, physical exercise also reduces blood glucose. It is therefore possible that the cognitive enhancing effects of physical exercise are mediated, at least in part,by the beneficial effect of lower glucose on the dentate gyrus.
Whether with physical exercise, diet or through the development of potential pharmacologic interventions, the research suggests that improving glucose metabolism could be a clinically viable approach for improving the cognitive slide that occurs in many people as they age.
With increasing longevity and the aging of the baby boom population, cognitive decline has emerged as a major health care crisis and concern. By being able to investigate patients and animal models in parallel studies, this new application will help researchers learn more about diseases of the brain.
This study was built upon an extensive, a series of ongoing epidemiological imaging study that evaluated 240 healthy elders at the Taub Institute for Research on Alzheimer's Disease and the Aging Brain at Columbia University Medical Center.
