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Thermogenic Weight Loss Herbal Amino TrimThermogenic Weight Loss

Herbal Amino Trim

$14.99 90 Count Bottle VP1861P Retails for $17.90
$149.99 12 Bottles 90 Count Each Retail Value $214.80

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Thermogenic Herbal Supplement with L-Phenylalanine Natural Weight Loss Formula

Proprietary Herbal Amino Trim supplies L-Phenylalanine, an essential amino acid plus powerful thermogenic herbal extracts, useful for weight loss diets ...developed as a natural alternative to prescription diet drugs

Herbal Amino Trim contains these key ingredients per 3 tablets:

No caffeine, corn, gluten, milk or egg derivatives, salt, sodium, soy, starch, sugar, wheat or yeast. No artificial colorings, flavorings or preservatives.

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Dietary Protein Promotes Thermogenesis For Effective Weight-Loss!

Metabolism works by converting food energy to other forms of energy to fuel muscle contraction, regulate fluids, conduct nerve impulses and build and repair tissues.

(Journal-American Coll Nutr, 23:373-385, 2004)

For a weight loss program to be safe and responsible, it should include these features:

NEW RESEARCH EXPLORES HOW EFFICIENT FAT BURNING "LIPOLYSIS" WORKS IN THE BODY

Researchers At The Albert Einstein College Of Medicine Shed Light On How The Fat Burning Process Works In The Body

Researchers have found that fat cells generate feedback to the brain in order to regulate fat burning in the body much the same way a thermostat regulates temperature inside a house.

With as increase in obesity threatening overall health and reducing life expectancies of people across the world, the research may help scientists better understand how weight is shed most effectively.

A team of researchers at the Albert Einstein College of Medicine, Georgia State University found that during the process of burning fat called lipolysis, fat cells use sensory nerves to feed information to the brain.

In their studies focusing on the transmission of information through the nervous system, the researchers found that the brain uses part of the nervous system used to regulate body functions, called the sympathetic nervous system. The system enables communication back to the cells to initiate, continue or stop the fat burning depending upon the information the brain receives from the fat.

The brain can actually trigger lipid burning by fat cells and through these sensory nerves, the fat cell can give the brain feedback. Understanding how this biological concept works to regulate the process of lipolysis is similar to how a thermostat regulates temperature in a house, using input from the air and output to a furnace or heating unit.

When the body has a low amount of a type of readily available fuel glycogen, a carbohydrate, the body starts lipolysis to release energy stored in fats. At the end of the communication process through the nerves which are part of the sympathetic nervous system, a naturally-occuring chemical called norepinephrine is released to trigger the breakdown of fat.

Sensory nerves then appear to report back to the brain to inform it of the status of the lipolysis, communicating whether too much or too little energy has been released, and the activity of the sympathetic nerves can be adjusted accordingly.

When there's a moderate amount of physical exercise or if there's been a fairly long interval since eating , a person will use up all or most of the available glycogen, necessitating the break down fat to yield sufficient energy. But you don't want to break down more than you need, so the process is able to stop the sympathetic nervous system from triggering the release of too much lipid energy from fat.

The researchers caution... Although this communication process is known to play a role in the short-term burning of fat, researchers are not certain whether this process is involved with the long-term issues of burning fat. It's crucial to understanding obesity and why some people burn fat more readily than others.

They theorize it could be that sensory nerves perform a dual function. In addition to the moment-to-moment lipolysis process, they might also have a longer term function. It's complex process, leading them to believe it might be a different subset of the sensory nerves performing the long-term monitoring of fat reserves.

The research appears in the March edition of the American Journal of Physiology: Regulatory, Integrative and Comparative Physiology.


How Exercise Minimizes Weight Regain...

By Reducing Appetite And Burning Fat First, Storing Carbohydrates For Use Later, Exercise Actually Helps Minimize Overeating.

According to a new study, exercise helps prevent weight regain after dieting by reducing appetite and by burning fat before burning carbohydrates. Burning fat first and storing carbohydrates for use later in the day slows weight regain and may actually minimize overeating by signaling a feeling of "fullness" to the brain.

The University of Colorado Denver study also found that exercise prevents the increase in the number of fat cells that occurs during weight regain, challenging prior conventional wisdom that the number of fat cells is set and cannot be altered by dietary or lifestyle changes.

These coordinated physiological changes in the brain and the body lower the weight that our physiology drives people to achieve, and suggest that the effects of exercise on these physiological processes may make it easier to stay on a diet.

The study is "Regular exercise attenuates the metabolic drive to regain weight after long term weight loss." conducted by a team of researchers of the University of Colorado Denver. The American Physiological Society published the research in the American Journal of Physiology - Regulatory, Integrative and Comparative Physiology.

How Exercise Works...

Weight gain is, on the surface, remarkably simple, occurring when the calories consumed exceeds the calories expended. On closer examination, the process is remarkably complex. Laboratory animals eat according to physiological signals that may suppress appetite or arouse the desire to eat. These signals are relatively weak in humans, as their intake is largely influenced by psychological, cognitive and lifestyle factors. After dieting, however, the physiological signals emerge to play a more substantial role in controlling intake. Being persistently hungry after losing weight with restricted diets is a big part of the weight regain problem. Most people are unable to ignore this physiological cue and are pushed by their biology to overeat and regain the weight they worked so hard to lose.

Some people are successful at keeping the weight off, and those tracked by The National Weight Control Registry share a number of common characteristics, including a program of regular exercise. The aim of this investigation was to uncover how exercise affects the body's physiology to minimize weight regain.

The researchers used obesity-prone laboratory subjects. For the first 16 weeks, they ate a high-fat diet, as much as they wanted, and remained sedentary. They were then placed on a diet. For the following two weeks, the subjects ate a low-fat and low-calorie diet, losing about 14% of their body weight. They maintained the weight loss by dieting for eight more weeks. Half the subjects exercised regularly on a treadmill during this period while the other half remained sedentary.

In the final 8-weeks, the relapse phase of the study, the subjects stopped dieting and ate as much low-fat food as they wanted. The exercise group continued to exercise and the sedentary group remained sedentary.

Compared to the sedentary group, the exercisers:

During feeding, the sedentary group preferentially burned carbohydrates while sending fat from the diet to fat tissue. This preferential fuel use stores more calories because it requires less energy to store fat than to store carbohydrates. In addition, burning away the body's carbohydrates may contribute to the persistent feeling of hunger and large appetite of the sedentary subjects.

Exercise blunted this fuel preference, favoring the burning of fat for energy needs and saving ingested carbohydrates so that they could be used later in the day. Taken together, the exercise led to a much lower appetite and fewer calories ending up in fat tissue.

The researchers also found that exercise prevented the increase in the number of fat cells observed with weight regain in sedentary subjects. In the sedentary group, a population of very small, presumably new, fat cells appears early in the relapse process. Small, new fat cells would not only accelerate the process of regain, but also increase fat storage capacity in the abdomen. It would also explain why sedentary subjects overshoot their previous weight when they relapse.

According to conventional wisdom, the number of fat cells is determined by genetics, rather than being regulated by diet or lifestyle. The team will do further research to demonstrate that exercise is, indeed, preventing the formation of new fat cells early in relapse and not simply altering the size of pre-existing fat cells.

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